Is Aspartame safe or not!

Read this article to learn why it is still being used despite numerous studies!

The video Monsanto does not want you to see!

Take the time to watch this!

Green Colon - An Unusual Occurrence?

Green Colons, Blue Colons read for yourself!

U.S. Department of Health and Human Services Finds Blue No. 2 to be TOXIC!

The title says it all so why are we still using it?

Do you know what these labels mean?

This handy guide will help your navigate your supermarket with confidence.

Monday, November 26, 2012

Kraft Remove Nitrates From Lunchables - Petition

Saturday, November 24, 2012

Vital Gatorade Petition Please View and Sign

Wednesday, November 21, 2012

Some links for the holiday weekend.

I don't like to post articles without scientific evidence backing them. I'd really like to get this information out there so I'm going to post a few links here for some holiday reading. As soon as I get a change after this upcoming weekend I will track down the abstracts and full texts if available for now enjoy the read and enjoy your holiday weekend!

MSG & Aspartame During Pregnancy

Artificial Sweeteners Linked to Weight Gain


Aspartame, AminoSweet, and Neotame

Monsanto canteen no longer serves GMO's!

Monsanto's Canteen In High Wycombe, Buckinghamshire No Longer Serves GMO's.

 We have probably all seen this story circulating around the web on various sites in various forms so what the heck I'll do an article on it as well. Monsanto the largest manufacturer of GMO seeds and of course Roundup, Agent Orange and a plethora of other deadly toxins does not serve it's own employees the GMO food it claims to be safe. This story actually surfaced in 1999 originally and is in regards to the pharmaceutical factory at High Wycombe, Buckinghamshire when it was verified by Monsanto spokesman Tony Coombes "Yes, this is the case, and it is because we believe in choice," Do I get a choice I wonder? If they believe in choice why are they so opposed to labeling foods that contain GMO's?

 What is obviously compelling is the fact that Sutcliffe Catering who actually provides and serves the food in the canteen stated the reason was: "to remove, as far as practicable, GM soya and maize from all food products served in our restaurant. We have taken the above steps to ensure that you, the customer, can feel confident in the food we serve."  

Kind of strange that Monsanto employees must have had to complain that they were in fact not confident with the food they serve which would of course also mean they are not confident in eating GMO's. Monsanto spokesman Tony Coombes also stated employees at their agribusiness plant at Cambridge were happy to eat GMO's "The notice in the restaurant there says some products may contain GMOs because our staff are happy to eat food sprayed with fewer chemicals."
Well we know that is completely untrue take a look at this study. Regardless in my opinion that sign in Cambridge was there because the staff knows better than to eat that crap there's no way to verify the staff's opinion at either location since they are not allowed to discuss internal matters.

 Oh well at least Monsanto employee's are smart enough not to eat GMO's and they at least have the choice, to bad they won't stand up and just say that publicly or grow a pair and just all walk off the job instead of poisoning the world. I will reiterate this story was originally posted in 1999 who knows what other Monsanto locations are no longer serving GMO's since then.

 I will leave us with a quote for all the Monsanto employees out there: "To allow an atrocity is to commit one thyself"

Tuesday, November 13, 2012

Saccharin Leads to Weight Gain - Study

A Role for Sweet Taste: Calorie Predictive Relations in Energy Regulation by Rats

Authors: Susan E. Swithers and Terry L. Davidson
Purdue University


Animals may use sweet taste to predict the caloric contents of food. Eating sweet noncaloric substances
may degrade this predictive relationship, leading to positive energy balance through increased food intake
and/or diminished energy expenditure. These experiments were designed to test the hypothesis that
experiences that reduce the validity of sweet taste as a predictor of the caloric or nutritive consequences
of eating may contribute to deficits in the regulation of energy by reducing the ability of sweet-tasting
foods that contain calories to evoke physiological responses that underlie tight regulation. Adult male
Sprague–Dawley rats were given differential experience with a sweet taste that either predicted increased
caloric content (glucose) or did not predict increased calories (saccharin). We found that reducing the
correlation between sweet taste and the caloric content of foods using artificial sweeteners in rats resulted
in increased caloric intake, increased body weight, and increased adiposity, as well as diminished caloric
compensation and blunted thermic responses to sweet-tasting diets. These results suggest that consumption of products containing artificial sweeteners may lead to increased body weight and obesity by
interfering with fundamental homeostatic, physiological processes.

Full Text Available Here!

Olestra Fat Substitute Linked to Weight Gain causes dysregulation - Study

Fat Substitutes Promote Weight Gain in Rats Consuming High-Fat Diets

Authors: Susan E. Swithers, Sean B. Ogden, and Terry L. Davidson

Purdue University


The use of food products designed to mimic the sensory properties of sweet and fat while providing fewer
calories has been promoted as a method for reducing food intake and body weight. However, such products
may interfere with a learned relationship between the sensory properties of food and the caloric consequences
of consuming those foods. In the present experiment, we examined whether use of the fat substitute, olestra,
affect energy balance by comparing the effects of consuming high-fat, high-calorie potato chips to the effects
of consuming potato chips that sometimes signaled high calories (using high-fat potato chips) and that
sometimes signaled lower calories (using nonfat potato chips manufactured with the fat substitute olestra).
Food intake, body weight gain and adiposity were greater for rats that consumed both the high-calorie chips
and the low-calorie chips with olestra compared to rats that consumed consuming only the high-calorie chips,
but only if animals were also consuming a chow diet that was high in fat and calories. However, rats
previously exposed to both the high- and low-calorie chips exhibited increased body weight gain, food intake
and adiposity when they were subsequently provided with a high fat, high calorie chow diet suggesting that
experience with the chips containing olestra affected the ability to predict high calories based on the sensory
properties of fat. These results extend the generality of previous findings that interfering with a predictive
relationship between sensory properties of foods and calories may contribute to dysregulation of energy
balance, overweight and obesity.

Full Text Available Here!

Olestra, Vitamin Deficiencies and Gastro Issues

It should be stated here that Olestra was banned in Canada due to the fact that Procter and Gamble could not prove it was a safe product. Olestra is more commonly known as Olean.

Olestra: A New Food Additive


In 1987, Procter and Gamble Company (Cincinnati, Ohio) petitioned the US Food and Drug Administration (FDA) to amend the food additive regulations to allow sucrose esterified with fatty acids (olestra) to be used as a replacement for conventional fats. The petitioner later restricted its request for use in savory snacks. FDA considered evidence submitted by the petitioner, the opinions of experts, proceedings from the FDA Food Advisory Committee, and public discussion and concluded on January 25, 1996, that olestra was safe for use in savory snacks (eg, salty snacks such as potato chips, corn chips). Olestra is not toxic, carcinogenic, genotoxic, or teratogenic and is neither absorbed nor metabolized by the body, but may be associated with gastrointestinal tract symptoms such as cramping or loose stools. In addition, olestra affects the absorption of fat-soluble vitamins but does not affect the absorption of water-soluble nutrients. The petitioner's studies concluded that when olestra was consumed with foods containing vitamins A, D, E, or K, the fat substitute could have an effect on the absorption of these nutrients. Therefore, FDA is requiring that fat-soluble vitamins lost through absorption be added back to olestra as follows: 170 IU vitamin A per gram olestra, 12 IU vitamin D per gram olestra, 2.8 IU vitamin E per gram olestra, and 8 μg vitamin K per gram olestra. As part of the conditions of approval FDA is requiring that the food labels of products containing olestra disclose the vitamin compensation and the potential gastrointestinal effects. FDA is also requiring that further studies examining consumption patterns and the effects of olestra on human beings be conducted. J Am Diet Assoc. 1998;98:565–569.

Consumption of monosodium glutamate in relation to incidence of overweight in Chinese adults: China Health and Nutrition Survey

Consumption of monosodium glutamate in relation to incidence of overweight in Chinese adults: China Health and Nutrition Survey 

  • Barry Popkin

  • Abstract

    Background: It has been hypothesized that monosodium glutamate (MSG), a flavor enhancer, is positively associated with weight gain, which influences energy balance through the disruption of the hypothalamic signaling cascade of leptin action.
    Objective: The objective was to examine the longitudinal association between MSG consumption and incidence of overweight.
    Design: Data were collected from the China Health and Nutrition Survey (CHNS), a prospective open-cohort, ongoing nationwide health and nutrition survey, consisting of 10,095 apparently healthy Chinese adults aged 18–65 y at entry from 1991 to 2006. Diet, including MSG and other condiments, was assessed with a weighed food inventory in combination with three 24-h recalls. Incident overweight was defined as a body mass index (BMI; in kg/m2) ≥ 25 or ≥23 based on World Health Organization recommendations for Asian populations. Multilevel mixed-effects models were constructed to estimate change in BMI, and Cox regression models with gamma shared frailty were used to determine the incidence of overweight.
    Results: The mean follow-up was 5.5 y. The cumulative mean (±SD) MSG intake of 2.2 ± 1.6 g/d was positively associated with BMI after adjustment for potential confounders and cluster effects at different levels (individual, household, and community). The adjusted hazard ratio of overweight was 1.33 (95% CI: 1.01, 1.75; P for trend < 0.01) for participants in the highest quintile of MSG intake compared with those in the lowest quintile after adjustment for age, physical activity, total energy intake, and other major lifestyle factors.
    Conclusions: MSG consumption was positively, longitudinally associated with overweight development among apparently healthy Chinese adults. Additional studies are needed to elucidate mechanisms of action and to establish causal inference.
  • Long term toxicity of a Roundup herbicide and a Roundup-tolerant genetically modified maize

    Here is yet another study that shows... well basically were all being poisoned by Monsanto.

    a b s t r a c t
    The health effects of a Roundup-tolerant genetically modified maize (from 11% in the diet), cultivated
    with or without Roundup, and Roundup alone (from 0.1 ppb in water), were studied 2 years in rats. In
    females, all treated groups died 2–3 times more than controls, and more rapidly. This difference was visible in 3 male groups fed GMOs. All results were hormone and sex dependent, and the pathological pro-
    files were comparable. Females developed large mammary tumors almost always more often than and
    before controls, the pituitary was the second most disabled organ; the sex hormonal balance was modified by GMO and Roundup treatments. In treated males, liver congestions and necrosis were 2.5–5.5
    times higher. This pathology was confirmed by optic and transmission electron microscopy. Marked
    and severe kidney nephropathies were also generally 1.3–2.3 greater. Males presented 4 times more large
    palpable tumors than controls which occurred up to 600 days earlier. Biochemistry data confirmed very
    significant kidney chronic deficiencies; for all treatments and both sexes, 76% of the altered parameters
    were kidney related. These results can be explained by the non linear endocrine-disrupting effects of
    Roundup, but also by the overexpression of the transgene in the GMO and its metabolic consequences.
    ! 2012 Elsevier Ltd. All rights reserved

    Full Text Available Here!

    Thursday, November 8, 2012

    Russian National Academy of Sciences: Surov's Hamsters Study

    Ultrastructural analysis of testes from mice fed on genetically modified soybean.

    Authors: Vecchio l, Cisterna B, Malatesta M, Martin T.E., Biggiogera M

    We have considered the possible effects of a diet containing genetically modified (GM) 
    soybean on mouse testis. This organ, in fact, is a well known bioindicator and it has 
    already been utilized, for instance, to monitor pollution by heavy metals. In this 
    preliminary study, we have focussed our attention on Sertoli cells, spermatogonia and 
    spermatocytes by means of immunoelectron microscopy. Our results point out that the 
    immunolabelling for Sm antigen, hnRNPs, SC35 and RNA Polymerase II is decreased 
    in 2 and 5 month-old GM-fed mice, and is restored to normal at 8 months. In GM-fed 
    mice of all ages considered, the number of perichromatin granules is higher and the 
    nuclear pore density lower. Moreover, we found enlargements in the smooth 
    endoplasmic reticulum in GM-fed mice Sertoli cells. A possible role played by traces of 
    the herbicide to which the soybean is resistant is discussed. 

    Sorry no dice on locating the full text of this study this may help anyone interested in finding it.

    Published European journal of histochemistry : EJH
    Volume 48, Issue 4, October 2004, Pages 448-454